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Arrest of activation of the p65 NF-kB protein in the skin keratinocytes, by knockout of either p65 itself or of IKK2, the kinase mediating p65 NF-kB activation, also results in inflammatory skin disorder Hu et al. 1999, Pasparakis et al. 2002. In that case, the disorder was shown to be triggered by chronic autocrine stimulation of the keratinocytes by TNF and a resulting enhancement of Jun phosphorylation, which, in the absence of a restraining effect of co-activated p65 occurs to an excessive extent. To explore the relationship between this skin inflammatory process and that which occurs as a result of caspase-8 deficiency in the keratinocytes, we assessed the contribution of TNF to the latter. can you buy cytoxan in canada cytoxan

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